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Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


ESCHERICHIA COLI INFECTIONS

Fig. 1. Congestion (overfilling of blood vessels with red blood cells) of the liver as an initial manifestation of E. coli septicaemia in a broiler chicken. H/E, Bar = 40 µm.

Fig. 1. Congestion (overfilling of blood vessels with red blood cells) of the liver as an initial manifestation of E. coli septicaemia in a broiler chicken. H/E, Bar = 40 µm.

 
Fig. 2. Serofibrinous perihepatitis consequent to E. coli septicaemia in a broiler chicken. Huge fibrinous pseudomembrane (f), coating the liver surface. H/E, Bar = 50 µm

Fig. 2. Serofibrinous perihepatitis consequent to E. coli septicaemia in a broiler chicken. Huge fibrinous pseudomembrane (f), coating the liver surface. H/E, Bar = 50 µm

 
Fig. 3. Serofibrinous perihepatitis. Organization of pseudomembranous deposits. H/E, Bar = 50 µm.

Fig. 3. Serofibrinous perihepatitis. Organization of pseudomembranous deposits. H/E, Bar = 50 µm.

 
Fig. 4. Е. coli septicaemia. Massive perivascular liver necrosis. H/E, Bar = 30 µm.

Fig. 4. Е. coli septicaemia. Massive perivascular liver necrosis. H/E, Bar = 30 µm.

 
Fig. 5. Periarteriolar reaction, fibrinoid necrosis and congestion of the spleen in Е. coli septicaemia. H/E, Bar = 30 µm.

Fig. 5. Periarteriolar reaction, fibrinoid necrosis and congestion of the spleen in Е. coli septicaemia. H/E, Bar = 30 µm.

 
Fig. 6. Croupous pleuropneumonia - one of the commonest findings in Е. coli septicaemia of respiratory origin. H/E, Bar = 25 µm.

Fig. 6. Croupous pleuropneumonia - one of the commonest findings in Е. coli septicaemia of respiratory origin. H/E, Bar = 25 µm.

 
Fig. 7. Serofibrinous pericarditis and inflammatory oedema of the myocardium in Е. coli septicaemia. H/E, Bar = 30 µm.

Fig. 7. Serofibrinous pericarditis and inflammatory oedema of the myocardium in Е. coli septicaemia. H/E, Bar = 30 µm.

 
Fig. 8. Degenerative necrobiotic lesions in the epithelium of renal tubules. H/E, Bar = 25 µm.

Fig. 8. Degenerative necrobiotic lesions in the epithelium of renal tubules. H/E, Bar = 25 µm.

 
Fig. 9. Catarrhal haemorrhagic enteritis caused by an enterotoxigenic Е. coli strain. H/E, Bar = 100 µm.

Fig. 9. Catarrhal haemorrhagic enteritis caused by an enterotoxigenic Е. coli strain. H/E, Bar = 100 µm.

 
Fig. 10. Element of Fig. 9. Massive haemorrhages surrounding lymphatic clusters in the intestinal wall. H/E, Bar = 25 µm.

Fig. 10. Element of Fig. 9. Massive haemorrhages surrounding lymphatic clusters in the intestinal wall. H/E, Bar = 25 µm.

 
Fig. 11. Haemorrhages in the proventriculus mucous coat in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 100 µm.

Fig. 11. Haemorrhages in the proventriculus mucous coat in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 100 µm.

 
Fig. 12. Haemorrhages and cystic formations in the gizzard in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 40 µm.

Fig. 12. Haemorrhages and cystic formations in the gizzard in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 40 µm.

 
Fig. 13. Massive haemorrhages in the parenchyma of tonsila caecalis in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 50 µm.

Fig. 13. Massive haemorrhages in the parenchyma of tonsila caecalis in colisepticaemia of enteric origin, secondary to necrotic enteritis. H/E, Bar = 50 µm.

 
Fig 14. Coligranuloma (Hjarre’s disease). Conglomerates of granulomatous nodes against the intestinal wall. Central necrotic detritus and marked acidophilia. Single foreign body-type giant cells. H/E, Bar = 30 µm.

Fig 14. Coligranuloma (Hjarre’s disease). Conglomerates of granulomatous nodes against the intestinal wall. Central necrotic detritus and marked acidophilia. Single foreign body-type giant cells. H/E, Bar = 30 µm.

 
Fig 15. Coligranuloma (Hjarre’s disease). Coligranuloma, liver. H/E, Bar = 35 µm.

Fig 15. Coligranuloma (Hjarre’s disease). Coligranuloma, liver. H/E, Bar = 35 µm.

 
Fig. 16. Coligranuloma (Hjarre’s disease). Coligranuloma, liver. The typical heterophilic debris among the central necrotic detritus. H/E, Bar = 25 µm.

Fig. 16. Coligranuloma (Hjarre’s disease). Coligranuloma, liver. The typical heterophilic debris among the central necrotic detritus. H/E, Bar = 25 µm.

 
Fig. 17. Sternal bursitis. Filling of the sternal bursa with fibrinous caseous exudate. Peripheral outgrowth of fibrous tissue and inflammatory hyperaemia. H/E, Bar = 100 µm.

Fig. 17. Sternal bursitis. Filling of the sternal bursa with fibrinous caseous exudate. Peripheral outgrowth of fibrous tissue and inflammatory hyperaemia. H/E, Bar = 100 µm.

 
This book is protected by the copyright law. The reproduction, imitation or distribution of the book in whole or in part, in any format (electronic, photocopies etc.) without the prior consent, in writing, of copyright holders is strictly prohibited.

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