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Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


CHOLANGIOHEPATITIS

Fig 1. Liver. Multiple outgrown bile
ducts, forming a granuloma structure
with a central necrosis. H/E, Bar = 35
µm.

Fig 1. Liver. Multiple outgrown bile ducts, forming a granuloma structure with a central necrosis. H/E, Bar = 35 µm.

 
Fig. 2. Liver. A biliary stasis and a huge
amount of Gram-positive organisms in
bile ducts. Gram staining, Bar = 25 µm.

Fig. 2. Liver. A biliary stasis and a huge amount of Gram-positive organisms in bile ducts. Gram staining, Bar = 25 µm.

 
Fig. 3. Liver. Pericanalicular biliary necroses.
H/E, Bar = 50 µm.

Fig. 3. Liver. Pericanalicular biliary necroses. H/E, Bar = 50 µm.

 
Fig. 4. Liver. Fibrosed and thickened
wall of a bile duct, filled with Grampositive
microorganisms after the
occurred inflammatory and necrotic
changes. H/E, Bar = 35 µm.

Fig. 4. Liver. Fibrosed and thickened wall of a bile duct, filled with Grampositive microorganisms after the occurred inflammatory and necrotic changes. H/E, Bar = 35 µm.

 
Fig. 5. Hepatosis in the liver of a broiler
chicken. Proliferation of bile ducts
and fibrous tissue, that have infiltrated
the portal zones through mononuclear
cells. H/E, Bar = 25 µm.

Fig. 5. Hepatosis in the liver of a broiler chicken. Proliferation of bile ducts and fibrous tissue, that have infiltrated the portal zones through mononuclear cells. H/E, Bar = 25 µm.

 
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