converting website visitors

ThePoultrySite.com - news, features, articles and disease information for the poultry industry

Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


RICKETS

Fig. 1. Histopathological lesions in
rickets due to calcium and vitamin D
deficiency. The proliferative zone of
the growth plate is widened, irregular
and poorly vascularized. Only a
small zone of hypertrophic cartilage
and reduced calcification is present.
H/E, Bar = 40 µm.

Fig. 1. Histopathological lesions in rickets due to calcium and vitamin D deficiency. The proliferative zone of the growth plate is widened, irregular and poorly vascularized. Only a small zone of hypertrophic cartilage and reduced calcification is present. H/E, Bar = 40 µm.

 
Fig. 2. Histopathological lesions in rickets due to phosphate
deficiency or excessive calcium levels. The hypertrophic
zone (zh) of the growth plate is increased,
noncalcified, but normally vascularized by metaphyseal
blood vessels. H/E, Bar = 40 µm.

Fig. 2. Histopathological lesions in rickets due to phosphate deficiency or excessive calcium levels. The hypertrophic zone (zh) of the growth plate is increased, noncalcified, but normally vascularized by metaphyseal blood vessels. H/E, Bar = 40 µm.

 
Fig. 3. A hypertrophic zone, consisting
of multiple nonmineralized trabeculae
of cartilage in a case of vitamin
D deficiency. H/E, Bar = 35 µm.

Fig. 3. A hypertrophic zone, consisting of multiple nonmineralized trabeculae of cartilage in a case of vitamin D deficiency. H/E, Bar = 35 µm.

 
Fig. 4. In cases of prolonged calcium
deficiency, a removal of calcium from
the skeleton does occur. Consequently,
thinning of cortices of long bones is
resulting. Longitudinal cross-section,
femurs, 40-day-old broiler chickens.
Left: normal thickness of the bone
wall (n). Right: extreme thinning of
the cortex (r) due to demineralization
in a broiler chicken. H/E, Bar =
50 µm.

Fig. 4. In cases of prolonged calcium deficiency, a removal of calcium from the skeleton does occur. Consequently, thinning of cortices of long bones is resulting. Longitudinal cross-section, femurs, 40-day-old broiler chickens. Left: normal thickness of the bone wall (n). Right: extreme thinning of the cortex (r) due to demineralization in a broiler chicken. H/E, Bar = 50 µm.

 
Fig. 5. Sagittal cross-section of the
proximal femur in a broiler chicken,
case of calcium deficiency. Replacement
of bone substance by a newly
grown fibrous (f) tissue. H/E, Bar =
30 µm.

Fig. 5. Sagittal cross-section of the proximal femur in a broiler chicken, case of calcium deficiency. Replacement of bone substance by a newly grown fibrous (f) tissue. H/E, Bar = 30 µm.

 
Fig. 6. The detection of osteoclasts
(arrows) in some cases of calcium
deficiency rickets resembles the findings
in osteoporosis of laying hens.
H/E, Bar = 25 µm.

Fig. 6. The detection of osteoclasts (arrows) in some cases of calcium deficiency rickets resembles the findings in osteoporosis of laying hens. H/E, Bar = 25 µm.

 
Fig. 7. Tarsometatarsal bone, 35-dayold
broiler chicken, longitudinal crosssection.
Growth of subperiosteal
abnormal masses of hypertrophic
cartilage, resulting in extensive bone
thickening looking like osteopetrosis.
H/E, Bar = 35 µm.

Fig. 7. Tarsometatarsal bone, 35-dayold broiler chicken, longitudinal crosssection. Growth of subperiosteal abnormal masses of hypertrophic cartilage, resulting in extensive bone thickening looking like osteopetrosis. H/E, Bar = 35 µm.

 
This book is protected by the copyright law. The reproduction, imitation or distribution of the book in whole or in part, in any format (electronic, photocopies etc.) without the prior consent, in writing, of copyright holders is strictly prohibited.

Our Sponsors

Partners


Seasonal Picks

Poultry Breeds and Management