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Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


PULMONARY HYPERTENSION (ASCITES) SYNDROME IN BROILER CHICKENS

Fig. 1. Separation of intermyocardial
fibres by a moderate to extensive
oedema of loose connective tissue.
Subepicardial and myocardial haemorrhages
of a various size. H/E, Bar
= 50 µm.

Fig. 1. Separation of intermyocardial fibres by a moderate to extensive oedema of loose connective tissue. Subepicardial and myocardial haemorrhages of a various size. H/E, Bar = 50 µm.

 
Fig. 2. Lung. Hyperaemia, haemorrhages
and oedema. A possible finding
is the appearance of of bone and
cartilage foci (co) among the pulmonary
parenchyma. H/E, Bar = 30 µm.

Fig. 2. Lung. Hyperaemia, haemorrhages and oedema. A possible finding is the appearance of of bone and cartilage foci (co) among the pulmonary parenchyma. H/E, Bar = 30 µm.

 
Fig. 3. Liver sinusoids are enlarged
and overfilled with blood. Supplementary
hepatic findings are the
oedema and atrophic degenerative
lesions. H/E, Bar = 50 µm.

Fig. 3. Liver sinusoids are enlarged and overfilled with blood. Supplementary hepatic findings are the oedema and atrophic degenerative lesions. H/E, Bar = 50 µm.

 
Fig. 4. Compression atrophy of liver
parenchyma secondary to congested
sinusoids resulting from the passive
venous hyperaemia. H/E, Bar = 30
µm.

Fig. 4. Compression atrophy of liver parenchyma secondary to congested sinusoids resulting from the passive venous hyperaemia. H/E, Bar = 30 µm.

 
This book is protected by the copyright law. The reproduction, imitation or distribution of the book in whole or in part, in any format (electronic, photocopies etc.) without the prior consent, in writing, of copyright holders is strictly prohibited.

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